miércoles, 11 de junio de 2014

[OFER-TRABEC] NAC: Predoctoral PFIS and Postdoctoral Sara Borrell

Bolsa de empleo: http://www.biocat.cat/es/bolsa-de-empleo
(c)OFER-TRABEC es un servicio ofrecido por el Centro de Comunicaciones RedIRIS
(Permitida la reproducción siempre que se cite la fuente de este Servicio).

Contacto correo-e:mapujana@iconcologia.net
Predoctoral PFIS and Postdoctoral Sara Borrell candidates to be enrolled in Breast Cancer Research group (IDIBELL, Barcelona)

Deadline for applications is 20 June 2014.

We seek for:
- A highly motivated science master student with a BSc score > 2.5 (1-4).
- An excellent skilled postdoc with a strong publication record in cancer biology.

The student should apply to the Predoctoral Fellowship program of the Ministry of Health ISCIII (PFIS program), while we offer a 1 year renewable contract (starting 2015). "Contratos predoctorales de formacion en investigacion (PFIS)"

The postoc should apply to the Sara Borrell Fellowship program of the Ministry of Health ISCIII. Applicants must have first-authored publications in respected journals.

The candidates should be self-motivated and independent, and have strong training in biochemistry and cancer cell biology. The research group is situated within the Catalan Institute of Oncology (Bellvitge Institute for Biomedical Research, IDIBELL). The Institute offers strong interactive environment and various excellent opportunities for collaborative research exists within the institute and the surrounding Hospital area.

Interested applicants are encouraged to apply with an email containing your curriculum vitae and names and contact information of two referees to Miquel Àngel Pujana (email: mapujana@iconcologia.net).

Deadline for applications is 20 June 2014.

Información complementaria de la oferta:
Publications by the group:
Aguilar et al. (2014). VAV3 mediates resistance to breast cancer endocrine therapy. Breast Cancer Res. 2014 May 28;16(3):R53.
Bonifaci et al. (2014). Integrating gene expression and epidemiological data for the discovery of genetic interactions associated with cancer risk. Carcinogenesis. Jan 2., 2014.
Maxwell et al. (2011). Interplay between BRCA1 and RHAMM Regulates Epithelial Apicobasal Polarization and May Influence Risk of Breast Cancer. PLoS Biology Nov;9(11):e1001199.
Aguilar et al. (2010). Biological reprogramming in acquired resistance to endocrine treatment of breast cancer. Oncogene 29(45):6071-83.

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